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Fruit Anthracnose - Colletotrichum spp. |
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Fusarium Wilt - Fusarium oxysporum |
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Verticillium Wilt - Verticillium dahliae, V.
albo-atrum |
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Early Blight - Alternaria solani |
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Late Blight - Phytophthora infestans |
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Septoria Leaf Blight - Septoria lycopersici |
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Soil Rot of Fruit - Rhizoctonia |
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Pathogens: Fusarium oxysporum, Verticillium
dahliae, V. albo-atrum |
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Soil-borne pathogens |
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Survive in soil for several years |
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Plants generally infected through roots |
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Rotation (at least 2-3 years) |
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VF or VFN resistance |
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Pathogen:
Septoria lycopersici |
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Survives over winter on infected plant debris,
and also on equipment, stakes and cages |
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Spores dispersed by splashing water (rain,
irrigation), workers or equipment moving through wet plants |
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Favored by moist, warm weather |
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Symptoms generally appear first on lower leaves |
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Rotation (at least 1-2 years) |
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Thorough sanitation of equipment, stakes, etc. |
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Stake plants |
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Plastic mulches |
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Fungicide sprays |
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Several effective fungicides available |
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Apply at least weekly when weather favorable for
disease |
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Good coverage, especially of lower leaves,
crucial |
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Carefully read label directions |
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Pathogen:
Alternaria solani |
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Survives on infected plant debris - partially
buried debris is an excellent source of inoculum |
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Spores wind dispersed, can be carried long
distances |
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Greatest threat of infection: |
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Fields with short rotations |
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Planting adjacent to fields infected previous
year where debris not completely buried |
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Infection occurs first on oldest leaves |
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Plant rotation - minimum of at least 2-3 years
between solanaceous crops |
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Potato, tomato, weeds in nightshade family
excellent hosts |
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Completely bury plant debris after harvest |
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Fungicide applications |
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Several effective fungicides available |
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Carefully read label directions |
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Provide adequate nitrogen |
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Avoid excessive irrigation |
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8Pathogen:
Phytophthora infestans |
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!Same fungus that causes late blight of potato |
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! Isolates may differ in ability to infect
potato and tomato |
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! Both mating types (A1 and A2) are now found
in WI |
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8 Symptoms: |
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! Lesions on leaves initially water-soaked
spots with irregular borders |
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! Lesions turn pale green, then brown to
almost black |
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! White fungal growth appears on underside of
leaves at border between necrotic and healthy tissue |
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! Petiole and stem symptoms similar lead to
rapid death of plant |
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8 Source of inoculum: |
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! Cull piles of infected potatoes or tomatoes |
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! Infected tomato transplants |
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! Infected plants in neighboring fields |
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! Hairy nightshade |
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8 Favored by rain, high relative humidity |
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Tomato Diseases Late Blight
Control
Strategies |
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8 Destroy cull piles, and weed hosts |
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8 Plant disease-free tomato transplants |
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8 Protectant fungicides several effective
fungicides available |
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Pathogen:
Several species including Colletotrichum coccodes, C. dematium and C. gloeosporioides |
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Favored by wet weather and warm temperatures |
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Survives in soil and plant debris several years |
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Spores splashed by rain or irrigation onto
foliage and fruit |
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Ripe or overripe fruit most susceptible,
symptoms progress rapidly |
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Rotation (at least 2 years) |
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Weed control (eliminates alternate hosts) |
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Stake plants |
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Mulches |
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Optimum plant spacing |
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Remove and destroy infected fruit |
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Fungicide sprays |
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Fungicide sprays |
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Several effective fungicides available |
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Start when first fruit reaches size of a quarter |
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Continue weekly until harvest |
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Read labels carefully |
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Examples of label requirements: |
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rate of application |
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seasonal use limitations - amount / acre /
season |
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pre-harvest intervals |
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crops that can be grown in treated area next
year |
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Pathogens: |
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Bacterial speck: Pseudomonas
syringae pv. tomato |
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Bacterial spot: Xanthomonas
campestris pv. vesicatoria |
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Favored by high humidity, rainfall/irrigation |
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Pathogens seed borne |
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Dispersal - wind driven rain, clipping plants,
equipment moving between wet plants |
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Optimum temperature: |
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Bacterial speck: 65-75° F Bacterial Spot: 75-85° F |
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Clean transplants, pathogen-free seed |
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Plant rotation - at least 1-2 years between
solanaceous crops |
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Thoroughly incorporate plant debris after
harvest |
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Application of bactericides |
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Fixed coppers - alone or in combination with
fungicides |
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Heavy rain, wind-driven rain, hail decrease
efficacy |
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Pathogen:
Rhizoctonia solani |
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Symptoms: |
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Rotted tissue showing concentric bands of
alternating dark and light brown |
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Initially, rotted area is firm but becomes mushy
with invasion of soft rotting organisms |
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Affected areas eventually turn black |
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Overwinters in soil |
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Fungus enters tomato where fruit contacts soil |
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Symptoms may not be evident at harvest but can
develop in storage or shipment |
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mulching around base of plants to avoid fruit
contact with soil helps prevent infection |
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mulches include straw, grass clippings, paper,
plastic |
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staking or caging of plants helps to avoid fruit
contact with soil |
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Pathogen: Tobacco Mosaic Virus |
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Symptoms: |
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Light and dark green mottled areas on leaves |
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Necrosis of fruit with some strains |
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Stunting of plants depending on growth stage at
infection |
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Virus easily transmitted by workers, machinery |
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TMV may be present in tobacco products |
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Virus survives on equipment, in plant debris |
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Crop rotation minimum of 3 years |
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Workers should wash hands in detergent before
handling plants |
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Sanitize pruning equipment periodically |
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Use cultivars with TMV resistance |
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Pathogen: Cucumber Mosaic Virus |
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Symptoms - foliar: |
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Mosaic or mottle similar to symptoms produced by
TMV |
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Shoestring appearance of leaves (filiform or
threadlike) |
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General stunting of the plant |
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Symptoms fruit: |
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Yield reduction amount and size of fruit |
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Fruit often misshapen |
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Delay in maturity |
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Numerous strains of the virus exist many are
specific to tomato |
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Over 700 plant species are host to this virus |
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Usually spread by aphids |
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Eliminate weed hosts such as |
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Milkweed |
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Chickweed |
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Ragweed |
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Clover |
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White cockle |
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Carpet weed |
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Plant a non-host barrier (such as corn) around
the tomato crop |
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